Author
LEAL-BERTIOLI, SORAYA - Embrapa | |
MORETZOHN, MARCIO - Embrapa | |
OZIAS-AKINS, PEGGY - University Of Georgia | |
CHU, YE - University Of Georgia | |
Holbrook, Carl - Corley | |
BALLEN, CAROLINA - Embrapa | |
CHAVARRO, CAROLINA - University Of Georgia | |
ABERNATHY, BRIAN - Embrapa | |
PEARL, STEPHANIE - Embrapa | |
GODOY, IGNACIO - Agronomical Institute Of Campinas (IAC) | |
JACKSON, SCOTT - University Of Georgia | |
BERTIOLI, DAVID - University Of Brasilia |
Submitted to: Meeting Abstract
Publication Type: Abstract Only Publication Acceptance Date: 10/1/2014 Publication Date: N/A Citation: N/A Interpretive Summary: not required Technical Abstract: Diseases are important reducers of peanut (Arachis hypogaea) yield. Wild species generally harbor greater levels of resistance and even apparent immunity. Genomic regions confering resistance to foliar diseases and root knot nematodes have been identified in populations involving the wild progenitors of peanut, A. ipaensis and A duranensis crossed with species that harbor resistances to these pests: A. magna and A stenosperma. For introgression of these genomic regions, induced allotetraploids were produced: [A. gregoryi x A stenosperma]4x, [A. magna x A. stenosperma]4x and [A. batizocoi x A. stenosperma]4x and used to cross with elite varieties in Brazil and in the USA. Molecular markers residing in the vicinity of genomic regions controlling disease resistance were developed using the diploid peanut genome sequences to develop tightly linked, easy-to-use microsatellite and SNP markers for foreground selection. In addition to this, SNP markers were developed for monitoring introgression of wild species DNA into cultivated peanut on a genome scale by calling polymorphisms between the wild species and A. duranensis and A. ipaensis as proxies for the cultivated peanut component genomes. In this way, each genome component was dissected for marker development. These examples are among the first markers developed using the diploid peanut genomes. |