Author
Submitted to: Genbank
Publication Type: Other Publication Acceptance Date: 9/9/2016 Publication Date: N/A Citation: N/A Interpretive Summary: Technical Abstract: Maize white seedling 3 (w3) has served as a model albino-seedling mutant since its discovery in 1923. We show that the w3 phenotype is caused by disruptions in homogentisate solanesyl transferase (HST), an enzyme that catalyzes the committed step in plastoquinone-9 (PQ9) biosynthesis. This reaction lies at the heart of a complex metabolic network of plastidial antioxidants, pigments, and phytohormones. Disruption of PQ9 biosynthesis has broad-ranging implications, from nutritional quality of grains to plant defense. These transposons were identified as causal in three alleles of w3. These mutants are being used to further our knowledge of plant hormone metabolism surrounding environmental and biotic stresses. |