Skip to main content
ARS Home » Pacific West Area » Davis, California » Western Human Nutrition Research Center » Diet, Microbiome and Immunity Research » People & Locations » Shurong Huang
Shurong Huang
Diet, Microbiome and Immunity Research
Molecular Biologist

Phone: (530) 752-7376
Fax: (530) 752-5271

(Employee information on this page comes from the REE Directory. Please contact your front office staff to update the REE Directory.)


Publications (Clicking on the reprint icon Reprint Icon will take you to the publication reprint.)
Endotoxin may not be the major cause of postprandial inflammation in adults who consume a single high-fat or moderately high-fat meal - (Peer Reviewed Journal)
Temporal changes in postprandial blood transcriptomes reveal subject-specific pattern of expression of innate immunity genes after a high-fat meal Reprint Icon - (Peer Reviewed Journal)
Lemay, D.G., Huang, S., Huang, L., Alkan, Z., Kirschke, C.P., Burnett, D.J., Wang, Y., Hwang, D.H. 2019. Temporal changes in postprandial blood transcriptomes reveal subject-specific pattern of expression of innate immunity genes after a high-fat meal. Journal of Nutritional Biochemistry. 72:108209. https://doi.org/10.1016/j.jnutbio.2019.06.007.
Transgenic mice with ectopic expression of constitutively active TLR4 in adipose tissues do not show impaired insulin sensitivity - (Peer Reviewed Journal)
Ono-Moore, K.D., Zhao, L., Huang, S., Kim, J., Rutkowsky, J.M., Snodgrass, R.G., Schneider, D.A., Quon, M.J., Graham, J.L., Havel, P.J., Hwang, D.H. 2017. Transgenic mice with ectopic expression of constitutively active TLR4 in adipose tissues do not show impaired insulin sensitivity. European Journal of Immunology. doi: 10.1002/iid3.162.
What causes high fat diet-induced postprandial inflammation: endotoxin or free fatty acids? - (Abstract Only)
Docosahexaenoic acid and palmitic acid reciprocally modulate monocyte activation in part through endoplasmic reticulum stress - (Peer Reviewed Journal)
Snodgrass, R.G., Huang, S., Namgaladze, D., Jandali, O., Shao, T., Sama, S., Brune, B., Hwang, D.H. 2016. Docosahexaenoic acid and palmitic acid reciprocally modulate monocyte activation in part through endoplasmic reticulum stress. Journal of Lipid Research. doi: 10.1016/j.jnutbio.2016.01.010.
Acylcarnitines activate pro-inflammatory signaling pathways - (Peer Reviewed Journal)
Rutkowsky, J.M., Knotts, T.A., Ono-Moore, K.D., Mc Coin, C.S., Huang, S., Schneider, D.A., Singh, S., Adams, S.H., Hwang, D.H. 2014. Acylcarnitines activate pro-inflammatory signaling pathways. American Journal of Physiology - Endocrinology and Metabolism. DOI: 10.1152/ajpendo.00656.2013.
Saturated fatty acids activate TLR-mediated pro-inflammatory signaling pathways - (Peer Reviewed Journal)
Huang, S., Rutkowsky, J.M., Snodgrass, R.G., Ono-Moore, K., Schneider, D.A., Newman, J.W., Adams, S.H., Hwang, D.H. 2012. Saturated fatty acids activate TLR-mediated pro-inflammatory signaling pathways. Journal of Lipid Research. Epublished. DOI: 10.1194/jlr.D029546.
Inhibition of Nod2 signaling and target gene expression by curcumin - (Peer Reviewed Journal)
Huang, S., Zhao, L., Kim, K., Lee, D., Hwang, D.H. 2008. Inhibition of Nod2 signaling and target gene expression by curcumin. Molecular Pharmacology 74(1):274-281, 2008.