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ARS Home » People & Locations » Mary Briske Anderson
Mary J Briske Anderson
Grand Forks Human Nutrition Research Center
Microbiologist

Phone: (701) 795-8399
Fax: (701) 795-8395

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Publications (Clicking on the reprint icon Reprint Icon will take you to the publication reprint.)
Efficacy of butyrate to inhibit colonic cancer cell growth is cell type-specific and apoptosis-dependent Reprint Icon - (Peer Reviewed Journal)
Oncel, S., Safratowich, B.D., Lindlauf, J., Liu, Z., Palmer, D., Briske Anderson, M.J., Zeng, H. 2024. Efficacy of butyrate to inhibit colonic cancer cell growth is cell type-specific and apoptosis-dependent. Nutrients. 16(4). Article 529. https://doi.org/10.3390/nu16040529.
Deoxycholic acid modulates cell-junction gene expression and increases intestinal barrier dysfunction in Caco-2 cell monolayers Reprint Icon - (Peer Reviewed Journal)
Zeng, H., Safratowich, B.D., Cheng, W., Larson, K.J., Briske Anderson, M.J. 2022. Deoxycholic acid modulates cell-junction gene expression and increases intestinal barrier dysfunction in Caco-2 cell monolayers. Molecular Nutrition and Food Research. https://doi.org/10.3390/molecules27030723.
Methylselenol, a selenium metabolite, plays common and different roles in cancerous colon HCT116 cell and noncancerous NCM460 colon cell proliferation - (Peer Reviewed Journal)
Zeng, H., Briske Anderson, M.J., Wu, M., Moyer, M.P. 2012. Methylselenol, a selenium metabolite, plays common and different roles in cancerous colon HCT116 cell and noncancerous NCM460 colon cell proliferation. Nutrition and Cancer. 64(1):128-135.
Methylselenol, a selenium metabolite, plays a critical role in inhibiting colon cancer cell growth in vitro and in vivo - (Abstract Only)
Methylselenol, a selenium metabolite, plays a critical role in inhibiting colon cancer cell growth in vitro and in vivo - (Abstract Only)
Zeng, H., Briske Anderson, M.J. 2011. Methylselenol, a selenium metabolite, plays a critical role in inhibiting colon cancer cell growth in vitro and in vivo. Federation of American Societies for Experimental Biology Conference. 25:110.4.
The Influence of Copper (Cu) Deficiency in a Cardiomyocyte Cell Model (HL-1 Cell) of Ischemia/Reperfusion Injury - (Abstract Only)
Johnson, W.T., Briske Anderson, M.J., Curtis, S.A., Asher, E.T. 2010. The Influence of Copper (Cu) Deficiency in a Cardiomyocyte Cell Model (HL-1 Cell) of Ischemia/Reperfusion Injury. Journal of Federation of American Societies for Experimental Biology. 24:719.1.
Methylselenol, a Selenium Metabolite, Plays Common and Different Roles in Colonic Cancer and Nontumorigenic Colonic Cell Growth - (Abstract Only)
Zeng, H., Briske Anderson, M.J., Moyer, M.P. 2010. Methylselenol, a Selenium Metabolite, Plays Common and Different Roles in Colonic Cancer and Nontumorigenic Colonic Cell Growth. Journal of Federation of American Societies for Experimental Biology. 24:916.5.
Deoxycholic Acid and Selenium Metabolite Methylselenol Exert Common and Distinct Effects on Cell Cycle, Apoptosis, and MAP Kinase Pathway in HCT116 Human Colon Cancer Cells - (Peer Reviewed Journal)
Zeng, H., Botnen, J.H., Briske Anderson, M.J. 2010. Deoxycholic Acid and Selenium Metabolite Methylselenol Exert Common and Distinct Effects on Cell Cycle, Apoptosis, and MAP Kinase Pathway in HCT116 Human Colon Cancer Cells. Nutrition and Cancer. 62(1):85-92.
Deoxycholic acid and selenium metabolite methylselenol exert common and distinct effects on cell cycle, apoptosis, and MAP kinase pathway in HCT116 human colon cancer cells - (Abstract Only)
Zeng, H., Botnen, J.H., Briske Anderson, M.J. 2009. Deoxycholic acid and selenium metabolite methylselenol exert common and distinct effects on cell cycle, apoptosis, and MAP kinase pathway in HCT116 human colon cancer cells. Journal of Federation of American Societies for Experimental Biology. 23:338.8.
Differential effects of deoxycholic acid versus selenium metabolite methylselenol on cell cycle, apoptosis, and MAP kinase pathway in HCT116 human colon cancer cells - (Abstract Only)
Zeng, H., Botnen, J.H., Briske Anderson, M.J. 2009. Differential effects of deoxycholic acid versus selenium metabolite methylselenol on cell cycle, apoptosis, and MAP kinase pathway in HCT116 human colon cancer cells. American Association of Cancer Research Meeting. Published in Abstracts of Poster Presentations. No. 4.
THE SELENIUM METABOLITE METHYLSELENOL INHIBITS THE MIGRATION AND INVASION POTENTIAL OF HT1080 TUMOR CELLS - (Peer Reviewed Journal)
Zeng, H., Briske-Anderson, M.J., Idso, J.P., Hunt, C. 2006. The selenium metabolite methylselenol inhibits the migration and invasion potential of HT1080 tumor cells. Journal of Nutrition. 136:1528-1532.
SELENIUM METABOLITE METHYLSELENOL INHIBITS MIGRATION AND INVASION POTENTIAL OF HT1080 TUMOR CELLS - (Abstract Only)
Zeng, H., Briske Anderson, M.J., Idso, J.P., Hunt, C. 2006. Selenium metabolite methylselenol inhibits migration and invasion potential of HT1080 tumor cells [abstract]. FASEB J. 20(5):A1011.
INHIBITORY EFFECT OF PROLONGED-BUTYRATE TREATMENT ON MIGRATION AND INVASION OF HT1080 TUMOR CELLS - (Abstract Only)
Zeng, H., Briske-Anderson, M. 2005. Inhibitory effect of prolonged-butyrate treatment on migration and invasion of HT1080 tumor cells [abstract]. The Federation of American Societies for Experimental Biology Journal. 19(5):A1693.
PROLONGED BUTYRATE TREATMENT INHIBITS THE MIGRATION AND INVASION OF POTENTIAL HT1080 TUMOR CELLS - (Peer Reviewed Journal)
Zeng, H., Briske Anderson, M.J. 2005. Prolonged butyrate treatment inhibits the migration and invasion of potential HT1080 tumor cells. Journal of Nutrition. 135:291-295.
PRE-TREATMENT OF CACO-2 CELLS WITH ZINC DURING THE DIFFERENTIATION PHASE ALTERS THE KINETICS OF ZINC UPTAKE AND TRANSPORT - (Peer Reviewed Journal)
Reeves, P.G., Briske-Anderson, M.J., Johnson, L. 2001. Pre-treatment of caco-2 cells with zinc during the differentiation phase alters the kinetics of zinc uptake and transport. Journal of Nutritional Biochemistry. 12:674-684.
PHYSIOLOGIC CONCENTRATIONS OF MEDIA ZINC ALTER THE RELATIVE ABUNDANCE OF ATP7A MRNA AND PROTEIN IN CAC0-2 CELL - (Abstract Only)
Reeves, P.G., DeMars, L.C., Briske-Anderson, M.J. 2000. Physiologic concentrations of media zinc alter the relative abundance of ATP7A MRNA and protein in caco-2 cells [abstract]. The Federation of American Societies for Experimental Biology Journal. 14:A228.
PHYSIOLOGIC CHANGES IN ZINC CONCENTRATIONS OF CACO-2 CELLS ALTER THE UPTAKE AND TRANSPORT KINETICS OF ZINC - (Abstract Only)
ZINC-RELATED METALLOTHIONEIN METABOLISM IN BOVINE PULMONARY ARTERY ENDOTHELIAL CELLS - (Peer Reviewed Journal)
PHYSIOLOGIC CONCENTRATIONS OF ZINC AFFECT THE KINETICS OF COPPER UPTAKE ANDTRANSPORT IN THE HUMAN INTESTINAL CELL MODEL, CACO-2 - (Peer Reviewed Journal)
SUPPORT MEMBRANE POROSITY INFLUENCES THE MORPHOLOGICAL UNIFORMITY OF CACO-2CELLS IN CULTURE - (Abstract Only)
PHYSIOLOGIC CONCENTRATIONS OF ZINC AFFECT THE RATE AND KINETICS OF COPPER UPTAKE, TRANSPORT AND RELEASE IN CACO-2 CELLS - (Abstract Only)
THE INFLUENCE OF CULTURE CONDITIONS AND PASSAGE NUMBER ON THE MORPHOLOGY AND METABOLISM OF CACO-2 CELLS - (Peer Reviewed Journal)
THE EFFECT OF IN VITRO SUPPORT SYSTEMS ON THE MORPHOLOGICAL CHARACTERISTICSOF HUMAN COLON ADENOCARCINOMA AND CHORIOCARCINOMA CELLS - (Other)
COPPER UPTAKE AND TRANSPORT IN DIFFERENTIATED HUMAN COLON ADENOCARCINOMA CELLS AS AFFECTED BY HIGH-ZINC CONCENTRATIONS IN THE CULTURE MEDIA - (Peer Reviewed Journal)